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How to Increase HDL Cholesterol: What Works and by How Much

By Dr. Muhammad Awais Rabbani, MBBS, physician and US medical-billing operator · Medically reviewed by Dr. Ahmed Humayon, MBBS · Updated 2 November 2026 · Editorial policy

HDL is the “good” cholesterol; below 40 mg/dL in men or 50 mg/dL in women is low. Regular aerobic exercise, losing excess weight, quitting smoking and replacing trans fats and refined carbs with unsaturated fats raise it most.

HDL result reader and habit planner

Sex
Habits you will add

Cut-offs from NCEP ATP III. Expected rises are meta-analysis averages (Kodama 2007, Dattilo & Kris-Etherton 1992, Maeda 2003). This tool plans habits; it does not diagnose.

Jump to: Lever table · Do pills help? · Retesting

What is HDL cholesterol, and what level is too low?

HDL cholesterol is the cholesterol carried by high-density lipoprotein, the particle that moves excess cholesterol from artery walls and tissues back to the liver. That is why it is called good cholesterol. An HDL below 40 mg/dL in men or below 50 mg/dL in women counts as low HDL cholesterol (NCEP ATP III, Circulation 2002; Grundy et al., Circulation 2005).

Your HDLMenWomen
Lowunder 40 mg/dL (1.03 mmol/L)under 50 mg/dL (1.29 mmol/L)
Target40 or more50 or more
Linked with lower risk60 or more (1.55 mmol/L)60 or more

Its partner, low-density lipoprotein (LDL), is the bad cholesterol because it delivers cholesterol into artery walls and other blood vessels.

Labs outside the US report HDL levels in mmol/L. To convert, divide mg/dL by 38.67. ATP III treats 60 mg/dL or more as a factor that lowers risk.

What causes low HDL cholesterol?

Low HDL cholesterol usually has several causes working at the same time. Physical inactivity, excess body weight, smoking, high triglycerides, insulin resistance, a diet heavy in refined carbohydrates, some medicines and genes all lower it. Most of them can be changed (NCEP ATP III, Circulation 2002).

  • Lifestyle: little physical activity, smoking, mostly refined-carb calories
  • Metabolic: excess weight, metabolic syndrome, type 2 diabetes and raised blood sugar levels, high triglycerides
  • Medicines: beta-blockers, anabolic steroids, progestins
  • Genes: rare inherited conditions that keep HDL very low

Low HDL and raised triglyceride levels tend to travel together. In overweight adults, a triglyceride-to-HDL ratio of 3.0 or more (mg/dL units) identified insulin resistance in one study (McLaughlin et al., Ann Intern Med 2003). Treat it as a clue that needs a proper workup.

Which changes raise HDL the most?

Aerobic exercise, losing excess weight, quitting smoking and swapping trans fats and refined carbs for unsaturated fats raise HDL the most without medicine. Each adds a few mg/dL, and the effects stack. Raising HDL cholesterol this way needs no prescription.

LeverTypical HDL changeEffortSource
Aerobic exercise+2.5 mg/dL120+ minutes a weekKodama et al., Arch Intern Med 2007
Weight loss+0.35 mg/dL per kg lost, after weight stabilizesModerate to highDattilo & Kris-Etherton, Am J Clin Nutr 1992
Quitting smoking+4 mg/dLHighMaeda et al., Prev Med 200300110-5)
Unsaturated fat replacing refined carbs or trans fatsmall rise; trans fat lowers HDLLow to moderateMensink et al., Am J Clin Nutr 2003; Mensink & Katan, NEJM 1990
Alcohol, 30 g a day+4 mg/dLNot a reason to startRimm et al., BMJ 1999
Niacin or CETP inhibitorslarge risePrescriptionHeart attacks did not fall (see below)

Each figure is an average from a meta-analysis; your change depends on your starting point.

To increase HDL by about 10 mg/dL, stack levers: exercise (+2.5), losing 10 kg (+3.5) and quitting smoking (+4) add up to roughly 10. The planner above adds these averages to your own result.

Bar chart: average HDL rise of 2.5 mg/dL from aerobic exercise, 3.5 mg/dL from losing 10 kg, 4 mg/dL from quitting smoking, about 10 mg/dL from all three together

How much aerobic exercise raises HDL?

Regular aerobic exercise raised HDL cholesterol by about 2.5 mg/dL on average across a meta-analysis of randomized controlled trials, and the gain followed a dose-response pattern. The effect appeared once people reached roughly 120 minutes a week or 900 kcal burned (Kodama et al., Arch Intern Med 2007).

Brisk walking, cycling and swimming all count at moderate intensity or harder. The American Heart Association recommends at least 150 minutes of moderate-intensity physical activity a week, which clears that threshold.

Regular exercise does more than boost HDL; it also lowers blood pressure.

Does losing weight or quitting smoking raise HDL?

Yes. Losing excess body weight and quitting smoking both raise HDL without medicine, and they sit at the core of any plan to increase HDL cholesterol naturally. Expect a smaller number during active weight loss, because HDL tends to dip while you are losing and rise once your weight holds steady (Dattilo & Kris-Etherton, Am J Clin Nutr 1992).

Weight loss

HDL rose about 0.35 mg/dL per kilogram lost after weight stabilized, so losing 10 kg adds about 3.5 mg/dL.

Smoking

Quitting smoking raised HDL by about 4 mg/dL in a meta-analysis of cessation studies (Maeda et al., Prev Med 2003).

Alcohol

About 30 g of alcohol a day, roughly two US standard drinks, raised HDL by about 4 mg/dL in controlled studies (Rimm et al., BMJ 1999). The American Heart Association advises people who do not drink not to start.

Which fats and carbs should you swap?

Swap trans fats and refined carbohydrates for unsaturated fats such as olive oil, nuts and fish. Replacing carbohydrate with unsaturated fat raises HDL slightly and lowers LDL in the same move (Mensink et al., Am J Clin Nutr 2003). Trans fat lowers HDL and raises LDL (Mensink & Katan, NEJM 1990).

  • Fried foods and baked goods made with hydrogenated oil → food cooked in olive or canola oil
  • White bread and sugary cereal → whole grains
  • Butter and fatty meat → nuts, seeds and oily fish

Saturated fats raise HDL a little but raise LDL more, so healthy fats are the better trade (Mensink et al., Am J Clin Nutr 2003).

What foods help raise HDL?

No single food raises HDL much. Olive oil, nuts, fatty fish, avocado and high-fiber foods help as part of the swap above, inside a heart-healthy diet. Their bigger payoff is usually lower LDL, so judge them by your whole lipid panel and not by the HDL line alone.

  • Olive oil: use it where you would use butter
  • Nuts: a small handful in place of chips or sweets
  • Fatty fish such as salmon or sardines: omega-3 fatty acids lower triglycerides
  • Avocado: monounsaturated fat in place of saturated fat
  • Oats, beans and lentils: soluble fiber lowers LDL and does little for HDL

Eggs are not an HDL lever. In a meta-analysis of 17 randomized trials, eating more eggs raised LDL and the LDL-to-HDL ratio (Li et al., Nutrients 2020).

How fast can you raise HDL?

Expect a measurable change after two to three months of steady effort. The exercise trials in the Kodama meta-analysis ran for at least eight weeks, and weight loss lifts HDL only once weight stabilizes, so one month is usually too soon to tell whether increasing HDL is working.

To raise HDL levels faster, combine levers; no single habit moves HDL far.

Do pills that raise HDL protect your heart?

No. Niacin and CETP inhibitors raised HDL substantially in large outcome trials, yet niacin added to a statin did not reduce heart attack or stroke, and the CETP inhibitors either failed or gave a small benefit that tracked lower non-HDL cholesterol. The 2018 AHA/ACC cholesterol guideline recommends no drug to raise HDL (Grundy et al., Circulation 2019).

DrugHDL effectOutcomeTrial
Niacin added to a statinupno fewer cardiovascular eventsAIM-HIGH, NEJM 2011
Niacin plus laropiprantupno fewer major vascular events; more serious side effectsHPS2-THRIVE, NEJM 2014
Torcetrapib (CETP)large risemore deaths and cardiovascular eventsILLUMINATE, NEJM 2007
Dalcetrapib, evacetrapib (CETP)upno benefitdal-OUTCOMES, NEJM 2012; ACCELERATE, NEJM 2017
Anacetrapib (CETP)large risesmall drop in coronary events, linked to lower non-HDL cholesterolREVEAL, NEJM 2017

Genetics points the same way. In a Mendelian randomization study, people born with a gene variant that raises HDL had no lower risk of heart attack (Voight et al., Lancet 201260312-2)). Low HDL marks risk; pushing the number up with a pill does not remove that risk.

Why does HDL matter at all?

HDL matters mainly as a risk marker. Low HDL often signals a wider metabolic problem, such as insulin resistance or high triglycerides, that raises the risk of cardiovascular disease. What HDL particles do matters more than how much cholesterol they carry, and a standard lipid panel does not measure that function.

One measure of function is cholesterol efflux capacity, the ability of HDL to pull cholesterol out of cells in the artery wall. It predicted atherosclerosis independently of HDL cholesterol (Khera et al., NEJM 2011). HDL particles are built around apolipoprotein A-I, but particle tests remain mostly research tools. Risk assessment leans more on apoB, which counts the particles that clog arteries.

Very large HDL values are not always protective either (Madsen et al., Eur Heart J 2017).

How to retest and read your next lipid panel

Retest 8 to 12 weeks after you change your habits, ideally at the same lab and time of day. The 2018 AHA/ACC guideline uses a 4- to 12-week window to check response to lifestyle change or medicine, and it accepts a nonfasting lipid panel for most adults (Grundy et al., Circulation 2019).

Biological variation means a single HDL level can move by several percent between two draws with no change in habits, so read one result as a range (Smith et al., Clin Chem 1993).

Many US labs print the total cholesterol-to-HDL ratio; lower is better, and the lab’s reference range shows its cutoff. Your LDL level is usually the main treatment target, so read it next to the HDL line and what your triglyceride number means. Other routine blood tests, like creatinine, can come from the same draw.

TestResultUnitsReference
Total cholesterol212mg/dLunder 200
Triglycerides180mg/dLunder 150
HDL cholesterol38mg/dL40 or more
LDL cholesterol (calculated)138mg/dLunder 100
Total cholesterol/HDL ratio5.6noneset by the lab

Sample layout with invented values, not a real patient. Cut-offs are ATP III’s; your lab’s may differ.

Who pays for the repeat test. A lipid panel is billed as CPT 80061. Medicare covers cardiovascular screening blood tests, including a lipid panel, once every five years when billed with the screening diagnosis ICD-10 Z13.6 (CMS, 42 CFR 410.17). Repeat panels to monitor a known lipid disorder fall under NCD 190.23 and need a diagnosis that supports medical necessity. Private plans set their own frequency rules, so check yours before a short-interval retest.

See a clinician if HDL stays low after three months of steady change.

Quick answers

How do you increase HDL cholesterol in women?

The same levers work, but the low cutoff is 50 mg/dL instead of 40. Progestins can lower HDL (NCEP ATP III, Circulation 2002), so mention hormonal contraception when you review results.

Which is good cholesterol, HDL or LDL?

HDL is the good cholesterol and LDL is the bad one. For HDL, higher is better up to a point; for LDL cholesterol, lower is better.

Can you raise HDL naturally?

Yes. Aerobic exercise, weight loss, quitting smoking and the fat swap raise HDL without pills, and together they can add several mg/dL over about three months.

Sources

  • National Cholesterol Education Program Expert Panel. Third Report of the NCEP Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults (ATP III) final report. Circulation. 2002;106(25):3143–3421.
  • Grundy SM, Cleeman JI, Daniels SR, et al. Diagnosis and management of the metabolic syndrome: an AHA/NHLBI scientific statement. Circulation. 2005;112(17):2735–2752.
  • McLaughlin T, Abbasi F, Cheal K, et al. Use of metabolic markers to identify overweight individuals who are insulin resistant. Ann Intern Med. 2003;139(10):802–809.
  • Kodama S, Tanaka S, Saito K, et al. Effect of aerobic exercise training on serum levels of high-density lipoprotein cholesterol: a meta-analysis. Arch Intern Med. 2007;167(10):999–1008.
  • Dattilo AM, Kris-Etherton PM. Effects of weight reduction on blood lipids and lipoproteins: a meta-analysis. Am J Clin Nutr. 1992;56(2):320–328.
  • Maeda K, Noguchi Y, Fukui T. The effects of cessation from cigarette smoking on the lipid and lipoprotein profiles: a meta-analysis. Prev Med. 2003;37(4):283–290.
  • Mensink RP, Zock PL, Kester ADM, Katan MB. Effects of dietary fatty acids and carbohydrates on the ratio of serum total to HDL cholesterol and on serum lipids and apolipoproteins: a meta-analysis of 60 controlled trials. Am J Clin Nutr. 2003;77(5):1146–1155.
  • Mensink RP, Katan MB. Effect of dietary trans fatty acids on high-density and low-density lipoprotein cholesterol levels in healthy subjects. N Engl J Med. 1990;323(7):439–445.
  • Rimm EB, Williams P, Fosher K, Criqui M, Stampfer MJ. Moderate alcohol intake and lower risk of coronary heart disease: meta-analysis of effects on lipids and haemostatic factors. BMJ. 1999;319(7224):1523–1528.
  • Li MY, Chen JH, Chen C, Kang YN. Association between egg consumption and cholesterol concentration: a systematic review and meta-analysis of randomized controlled trials. Nutrients. 2020;12(7):1995. doi:10.3390/nu12071995
  • AIM-HIGH Investigators. Niacin in patients with low HDL cholesterol levels receiving intensive statin therapy. N Engl J Med. 2011;365(24):2255–2267.
  • HPS2-THRIVE Collaborative Group. Effects of extended-release niacin with laropiprant in high-risk patients. N Engl J Med. 2014;371(3):203–212.
  • Barter PJ, Caulfield M, Eriksson M, et al. Effects of torcetrapib in patients at high risk for coronary events (ILLUMINATE). N Engl J Med. 2007;357(21):2109–2122.
  • Schwartz GG, Olsson AG, Abt M, et al. Effects of dalcetrapib in patients with a recent acute coronary syndrome (dal-OUTCOMES). N Engl J Med. 2012;367(22):2089–2099.
  • Lincoff AM, Nicholls SJ, Riesmeyer JS, et al. Evacetrapib and cardiovascular outcomes in high-risk vascular disease (ACCELERATE). N Engl J Med. 2017;376(20):1933–1942.
  • HPS3/TIMI55–REVEAL Collaborative Group. Effects of anacetrapib in patients with atherosclerotic vascular disease. N Engl J Med. 2017;377(13):1217–1227.
  • Voight BF, Peloso GM, Orho-Melander M, et al. Plasma HDL cholesterol and risk of myocardial infarction: a mendelian randomisation study. Lancet. 2012;380(9841):572–580.
  • Khera AV, Cuchel M, de la Llera-Moya M, et al. Cholesterol efflux capacity, high-density lipoprotein function, and atherosclerosis. N Engl J Med. 2011;364(2):127–135.
  • Madsen CM, Varbo A, Nordestgaard BG. Extreme high high-density lipoprotein cholesterol is paradoxically associated with high mortality in men and women: two prospective cohort studies. Eur Heart J. 2017;38(32):2478–2486.
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  • Smith SJ, Cooper GR, Myers GL, Sampson EJ. Biological variability in concentrations of serum lipids: sources of variation among results from published studies and composite predicted values. Clin Chem. 1993;39(6):1012–1022.
  • Centers for Medicare & Medicaid Services. 42 CFR § 410.17, Cardiovascular disease screening tests; National Coverage Determination 190.23, Lipid Testing.